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Mohsin Haematology Academy
EHA 2026 Congress · Stockholm

AML at EHA 2026

A negative frontline FLT3 readout that confirms current practice, with menin inhibitors and early cellular therapy alongside.

Fact-checked 16 June 2026
NegativeConfirms practice

PASHA

HOVON156/AMLSG28-18 · NCT04027309 · 768 patients

  • Gilteritinib vs midostaurin with intensive chemotherapy, frontline FLT3-mutated AML
  • Overall survival not met: HR 1.02 (0.81 to 1.28), P=0.864
  • 48-month OS 59% vs 60%
  • Event-free and relapse-free survival trended in favour of gilteritinib

Why it matters: A second-generation FLT3 inhibitor did not improve survival over the first-generation standard, so midostaurin plus chemotherapy remains the frontline option.

In practiceSelected use

Menin inhibitors

Revumenib, ziftomenib

  • Revumenib and ziftomenib are already approved in relapsed/refractory AML (KMT2A-rearranged or NPM1-mutant)
  • Ziftomenib CR/CRh 21.4% in KOMET-001
  • Frontline and combination use remains investigational

Why it matters: Menin inhibition is established in selected relapsed disease; the open question is how early in the pathway to use it.

Early phasePhase 1a

REVSTAR-123

RevSTAR-123 / AVC-201 · NCT05949125 · LBA5007 · Wermke

  • Switchable allogeneic CD123 CAR-T, delivered off the shelf
  • Complete remissions in CD123-positive relapsed/refractory AML
  • No graft-versus-host disease and no ICANS reported
  • A continuous targeting module acts as an on/off safety switch

Why it matters: An off-the-shelf, controllable CAR-T platform for a disease where conventional CAR-T has struggled with on-target toxicity.

Not every biologically superior drug improves overall survival.PASHA: gilteritinib matched but did not beat midostaurin for OS.